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The combination of nterleukin-10-1082 and tumor necrosis factor alpha-308 or interleukin-6-174 genes polymorphisms suggests an association with susceptibility to Hashimoto's thyroiditis

dc.contributor.authorBaki, Merve
dc.contributor.authorOZDERYA, Aysenur
dc.contributor.authorKARADAG, Berrin
dc.contributor.authorUysal, Mujdat
dc.contributor.authorAkman, Fevziye E.
dc.contributor.authorDogru-Abbasoglu, Semra
dc.contributor.authorVural, Pervin
dc.date.accessioned2021-03-02T22:53:54Z
dc.date.available2021-03-02T22:53:54Z
dc.date.issued2012
dc.identifier.citationBaki M., Akman F. E. , Vural P., Dogru-Abbasoglu S., OZDERYA A., KARADAG B., Uysal M., "The combination of nterleukin-10-1082 and tumor necrosis factor alpha-308 or interleukin-6-174 genes polymorphisms suggests an association with susceptibility to Hashimoto's thyroiditis", INTERNATIONAL IMMUNOPHARMACOLOGY, cilt.12, sa.4, ss.543-546, 2012
dc.identifier.issn1567-5769
dc.identifier.otherav_0f4ff78b-1a10-4d74-9c53-ff7f0041f89e
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/15839
dc.identifier.urihttps://doi.org/10.1016/j.intimp.2012.02.003
dc.description.abstractBackground: The etiopathogenesis of Hashimoto's thyroiditis (HT) has not been clearly elucidated although the role of chronical inflammation and endothelial dysfunction has been established. The imbalance between pro- and anti-inflammatory cytokines may play a role in the etiology. The aim of the present study was to investigate whether cytokine gene polymorphisms are associated with HT, and to evaluate the relationship between genotypes and clinical/laboratory manifestation of HT.
dc.language.isoeng
dc.subjectİmmünoloji
dc.subjectEczacılık
dc.subjectTemel Eczacılık Bilimleri
dc.subjectYaşam Bilimleri
dc.subjectTemel Bilimler
dc.subjectFarmakoloji ve Toksikoloji
dc.subjectFARMAKOLOJİ VE ECZACILIK
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectSağlık Bilimleri
dc.titleThe combination of nterleukin-10-1082 and tumor necrosis factor alpha-308 or interleukin-6-174 genes polymorphisms suggests an association with susceptibility to Hashimoto's thyroiditis
dc.typeMakale
dc.relation.journalINTERNATIONAL IMMUNOPHARMACOLOGY
dc.contributor.departmentIstanbul Sisli Hamidiye Etfal Training & Research Hospital , ,
dc.identifier.volume12
dc.identifier.issue4
dc.identifier.startpage543
dc.identifier.endpage546
dc.contributor.firstauthorID44613


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