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Dental follicle mesenchymal stem cell administration ameliorates muscle weakness in MuSK-immunized mice.

dc.contributor.authorUlusoy, Canan Aysel
dc.contributor.authorTasli, Hatice
dc.contributor.authorAkkoc, Tunc
dc.contributor.authorKucukerden, Melike
dc.contributor.authorTuzun, Erdem
dc.contributor.authorZibandeh, Noushin
dc.contributor.authorYildirim, Selin
dc.contributor.authorTRAKAS, Nikolaos
dc.contributor.authorZISIMOPOULOU, Paraskevi
dc.contributor.authorTZARTOS, Socrates
dc.contributor.authorGoker, Kamil
dc.date.accessioned2021-03-05T15:00:03Z
dc.date.available2021-03-05T15:00:03Z
dc.identifier.citationUlusoy C. A. , Zibandeh N., Yildirim S., TRAKAS N., ZISIMOPOULOU P., Kucukerden M., Tasli H., TZARTOS S., Goker K., Tuzun E., et al., "Dental follicle mesenchymal stem cell administration ameliorates muscle weakness in MuSK-immunized mice.", Journal of neuroinflammation, cilt.12, ss.231, 2015
dc.identifier.issn1742-2094
dc.identifier.othervv_1032021
dc.identifier.otherav_b9dd65a0-353b-4c35-adb9-794d419549f1
dc.identifier.urihttp://hdl.handle.net/20.500.12627/123641
dc.identifier.urihttps://doi.org/10.1186/s12974-015-0451-0
dc.description.abstractBackground: Myasthenia gravis (MG) is an antibody-mediated autoimmune disease of the neuromuscular junction (NMJ), mostly associated with acetylcholine receptor (AChR) antibodies. Around 5-10 % of MG patients show antibodies to muscle-specific tyrosine kinase (MuSK). Mesenchymal stem cell (MSC) administration has been shown to ameliorate muscle weakness in the experimental autoimmune myasthenia gravis (EAMG) model induced by AChR immunization.
dc.language.isoeng
dc.subjectTemel Bilimler
dc.subjectYaşam Bilimleri
dc.subjectSinirbilim ve Davranış
dc.subjectNEUROSCIENCES
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectİmmünoloji
dc.titleDental follicle mesenchymal stem cell administration ameliorates muscle weakness in MuSK-immunized mice.
dc.typeMakale
dc.relation.journalJournal of neuroinflammation
dc.contributor.departmentMarmara Üniversitesi , ,
dc.identifier.volume12
dc.identifier.startpage231
dc.identifier.endpage231
dc.contributor.firstauthorID2303


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