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Coronary artery calcification and chronically decreased GFR in living kidney donors

dc.contributor.authorBolayirli, Murat
dc.contributor.authorAltiparmak, Mehmet R.
dc.contributor.authorAkman, Canan
dc.contributor.authorKahveci, Arzu
dc.contributor.authorApaydin, Suheyla
dc.contributor.authorSariyar, Muzaffer
dc.contributor.authorSeyahi, Nurhan
dc.contributor.authorErek, Ekrem
dc.contributor.authorSerdengecti, Kamil
dc.contributor.authorAtaman, Rezzan
dc.date.accessioned2021-03-06T09:33:01Z
dc.date.available2021-03-06T09:33:01Z
dc.date.issued2007
dc.identifier.citationSeyahi N., Kahveci A., Bolayirli M., Akman C., Altiparmak M. R. , Apaydin S., Ataman R., Sariyar M., Serdengecti K., Erek E., "Coronary artery calcification and chronically decreased GFR in living kidney donors", AMERICAN JOURNAL OF KIDNEY DISEASES, cilt.49, ss.143-152, 2007
dc.identifier.issn0272-6386
dc.identifier.othervv_1032021
dc.identifier.otherav_e60a6d4c-d12d-45bf-b896-17b750bff211
dc.identifier.urihttp://hdl.handle.net/20.500.12627/151333
dc.identifier.urihttps://doi.org/10.1053/j.ajkd.2006.10.016
dc.description.abstractBackground: In the presence of decreased glomerular filtration rate (GFR), the risk of morbidity and mortality caused by cardiovascular disease (CVD) is increased markedly. Increased coronary artery calcification (CAC) is proposed as a pathogenetic link between CVD and chronic kidney disease. We examined the frequency and severity of CAC in living kidney donors to test the hypothesis that decreased GFR is associated with increased CAC.
dc.language.isoeng
dc.subjectSağlık Bilimleri
dc.subjectNefroloji
dc.subjectİç Hastalıkları
dc.subjectDahili Tıp Bilimleri
dc.subjectÜROLOJİ VE NEFROLOJİ
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.subjectTıp
dc.titleCoronary artery calcification and chronically decreased GFR in living kidney donors
dc.typeMakale
dc.relation.journalAMERICAN JOURNAL OF KIDNEY DISEASES
dc.contributor.department, ,
dc.identifier.volume49
dc.identifier.issue1
dc.identifier.startpage143
dc.identifier.endpage152
dc.contributor.firstauthorID180833


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