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The association between factor XIII Val34Leu polymorphism and early myocardial infarction

dc.contributor.authorOzben, B
dc.contributor.authorNalcaci, M
dc.contributor.authorBilge, AK
dc.contributor.authorOncul, A
dc.contributor.authorErgen, G
dc.contributor.authorHancer, VS
dc.contributor.authorDiz-Kucukkaya, R
dc.date.accessioned2021-03-06T20:14:00Z
dc.date.available2021-03-06T20:14:00Z
dc.date.issued2006
dc.identifier.citationHancer V., Diz-Kucukkaya R., Bilge A., Ozben B., Oncul A., Ergen G., Nalcaci M., "The association between factor XIII Val34Leu polymorphism and early myocardial infarction", CIRCULATION JOURNAL, cilt.70, ss.239-242, 2006
dc.identifier.issn1346-9843
dc.identifier.otherav_f95002aa-fd22-4c3e-9888-18666a09d06d
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/163297
dc.identifier.urihttps://doi.org/10.1253/circj.70.239
dc.description.abstractBackground Activated factor XIII (FXIII) cross-links between fibrin monomers, thus increasing the clot stability and resistance to fibrinolysis. Congenital FXIII deficiency causes severe bleeding diathesis. Recently, a common polymorphism of the FXIII A subunit (FXIII Val34Leu) has been identified as a protective factor against both arterial and venous thrombosis. The aim of this study was to investigate the role of FXIII Val34Leu polymorphism in coronary artery thrombosis, especially in young patients.
dc.language.isoeng
dc.subjectKardiyoloji
dc.subjectDahili Tıp Bilimleri
dc.subjectSağlık Bilimleri
dc.subjectTıp
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectCARDIAC ve CARDIOVASCULAR SİSTEMLER
dc.titleThe association between factor XIII Val34Leu polymorphism and early myocardial infarction
dc.typeMakale
dc.relation.journalCIRCULATION JOURNAL
dc.contributor.department, ,
dc.identifier.volume70
dc.identifier.issue3
dc.identifier.startpage239
dc.identifier.endpage242
dc.contributor.firstauthorID178115


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