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p53 but not p16(INK4a) induces growth arrest in retinoblastoma-deficient hepatocellular carcinoma cells

dc.contributor.authorÇAĞATAY, Tülin
dc.contributor.authorOzturk, M
dc.contributor.authorUnsal, K
dc.contributor.authorPonchel, F
dc.contributor.authorCarr, B
dc.contributor.authorMorel, AP
dc.date.accessioned2022-02-18T11:25:37Z
dc.date.available2022-02-18T11:25:37Z
dc.date.issued2000
dc.identifier.citationMorel A., Unsal K., ÇAĞATAY T., Ponchel F., Carr B., Ozturk M., "p53 but not p16(INK4a) induces growth arrest in retinoblastoma-deficient hepatocellular carcinoma cells", JOURNAL OF HEPATOLOGY, cilt.33, sa.2, ss.254-265, 2000
dc.identifier.issn0168-8278
dc.identifier.otherav_f7048eca-235c-4f39-9a8b-7cb377df3eae
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/181175
dc.identifier.urihttps://doi.org/10.1016/s0168-8278(00)80366-9
dc.description.abstractBackground/Aim: Both p16(INK4a) and p53 proteins are negative regulators of the cell cycle. In human hepatocellular carcinomas (HCC), the loss of function of p53, retinoblastoma (pRb) and p16(INK4a) genes by different mechanisms has been largely documented, but their hepatocellular effects are poorly known, We compared the growth-inhibitory effects of p16(INK4a) and p53 proteins in Hep3B cell line-derived clones.
dc.language.isoeng
dc.subjectGastroenterology
dc.subjectHepatology
dc.subjectHealth Sciences
dc.subjectDahili Tıp Bilimleri
dc.subjectGastroenteroloji-(Hepatoloji)
dc.subjectİç Hastalıkları
dc.subjectSağlık Bilimleri
dc.subjectTıp
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectGASTROENTEROLOJİ VE HEPATOLOJİ
dc.titlep53 but not p16(INK4a) induces growth arrest in retinoblastoma-deficient hepatocellular carcinoma cells
dc.typeMakale
dc.relation.journalJOURNAL OF HEPATOLOGY
dc.contributor.department, ,
dc.identifier.volume33
dc.identifier.issue2
dc.identifier.startpage254
dc.identifier.endpage265
dc.contributor.firstauthorID3371837


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