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dc.contributor.authorKitadai, Y
dc.contributor.authorChayama, K
dc.contributor.authorIto, M
dc.contributor.authorYasui, W
dc.contributor.authorHaruma, K
dc.contributor.authorHiyama, T
dc.contributor.authorTanaka, S
dc.contributor.authorYoshihara, M
dc.contributor.authorSasao, S
dc.contributor.authorKose, K
dc.contributor.authorShima, H
dc.contributor.authorTuncel, H
dc.contributor.authorUeno, Y
dc.date.accessioned2021-03-03T08:28:29Z
dc.date.available2021-03-03T08:28:29Z
dc.date.issued2004
dc.identifier.citationHiyama T., Tanaka S., Yoshihara M., Sasao S., Kose K., Shima H., Tuncel H., Ueno Y., Ito M., Kitadai Y., et al., "Chromosomal and microsatellite instability in sporadic gastric cancer", JOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY, cilt.19, sa.7, ss.756-760, 2004
dc.identifier.issn0815-9319
dc.identifier.othervv_1032021
dc.identifier.otherav_17be1c39-0dac-4bf6-87ea-d2b9efe56de9
dc.identifier.urihttp://hdl.handle.net/20.500.12627/21301
dc.identifier.urihttps://doi.org/10.1111/j.1440-1746.2004.03369.x
dc.description.abstractBackground: Gastric cancer can progress through two pathways of genomic instability: chromosomal (CIN) and microsatellite instability (MSI). It is hypothesized that these two pathways are not always independent and that some tumors show overlap between these two mechanisms.
dc.language.isoeng
dc.subjectSağlık Bilimleri
dc.subjectİç Hastalıkları
dc.subjectGastroenteroloji-(Hepatoloji)
dc.subjectDahili Tıp Bilimleri
dc.subjectTıp
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectGASTROENTEROLOJİ VE HEPATOLOJİ
dc.titleChromosomal and microsatellite instability in sporadic gastric cancer
dc.typeMakale
dc.relation.journalJOURNAL OF GASTROENTEROLOGY AND HEPATOLOGY
dc.contributor.department, ,
dc.identifier.volume19
dc.identifier.issue7
dc.identifier.startpage756
dc.identifier.endpage760
dc.contributor.firstauthorID172103


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