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dc.contributor.authorKucukoglu, M. Serdar
dc.contributor.authorBaskurt, Murat
dc.date.accessioned2021-03-03T10:10:02Z
dc.date.available2021-03-03T10:10:02Z
dc.identifier.citationBaskurt M., Kucukoglu M. S. , "Prostanoids in the treatment of pulmonary arterial hypertension", ANATOLIAN JOURNAL OF CARDIOLOGY, cilt.10, ss.2-8, 2010
dc.identifier.issn2149-2263
dc.identifier.othervv_1032021
dc.identifier.otherav_21105258-bcde-4476-b3e6-216e70aa95c8
dc.identifier.urihttp://hdl.handle.net/20.500.12627/27276
dc.identifier.urihttps://doi.org/10.5152/akd.2010.123
dc.description.abstractProstacyclin, endothelin-1, and nitric oxide pathways are involved in the pathogenesis of pulmonary arterial hypertension. This devastating disease of the pulmonary vasculature is associated with vasoconstriction, thrombosis and proliferation, and this may be partly due to lack of endogenous prostacyclin secondary to prostacyclin synthase downregulation. Prostanoids (prostacycin analogues) are potent vasodilators and possess antiaggregant, antiinflammatory and antiproliferative properties. The first agent to be approved for the treatment of pulmonary arterial hypertension was epoprostenol. In the last decade other prostanoids (treprostinil, iloprost) has been approved for the treatment of pulmonary arterial hypertension. (Anadolu Kardiyol Derg 2010; 10: Suppl 2; 2-8)
dc.language.isoeng
dc.subjectSağlık Bilimleri
dc.subjectKardiyoloji
dc.subjectDahili Tıp Bilimleri
dc.subjectTıp
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectCARDIAC ve CARDIOVASCULAR SİSTEMLER
dc.titleProstanoids in the treatment of pulmonary arterial hypertension
dc.typeMakale
dc.relation.journalANATOLIAN JOURNAL OF CARDIOLOGY
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume10
dc.identifier.startpage2
dc.identifier.endpage8
dc.contributor.firstauthorID197174


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