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dc.contributor.authorBarut, Kenan
dc.contributor.authorOzman, Sezen
dc.contributor.authorKasapcopur, Ozgur
dc.contributor.authorBatar, Bahadir
dc.contributor.authorGuven, Mehmet
dc.date.accessioned2021-03-03T14:45:28Z
dc.date.available2021-03-03T14:45:28Z
dc.date.issued2017
dc.identifier.citationBatar B., Ozman S., Barut K., Kasapcopur O., Guven M., "TNF-alpha 863C > A promoter and TNFRII 196T > G exonic variations may be risk factors for juvenile idiopathic arthritis", TURKISH JOURNAL OF MEDICAL SCIENCES, cilt.47, sa.6, ss.1819-1825, 2017
dc.identifier.issn1300-0144
dc.identifier.othervv_1032021
dc.identifier.otherav_3b44945a-e999-4c37-a608-bbfa2bbf740d
dc.identifier.urihttp://hdl.handle.net/20.500.12627/43813
dc.identifier.urihttps://doi.org/10.3906/sag-1612-23
dc.description.abstractBackground/aim: Juvenile idiopathic arthritis (JIA) is a chronic complex autoimmune disease. Genetic and environmental factors increase the risk of JIA. It is accepted that alterations in immune system pathways play an important role in the pathogenesis of JIA. The aim of the study was to investigate the possible association between immune system regulatory gene polymorphisms and JIA in Turkish patients.
dc.language.isoeng
dc.subjectKlinik Tıp (MED)
dc.subjectTIP, GENEL & İÇECEK
dc.subjectKlinik Tıp
dc.subjectTıp
dc.subjectSağlık Bilimleri
dc.subjectTemel Tıp Bilimleri
dc.titleTNF-alpha 863C > A promoter and TNFRII 196T > G exonic variations may be risk factors for juvenile idiopathic arthritis
dc.typeMakale
dc.relation.journalTURKISH JOURNAL OF MEDICAL SCIENCES
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume47
dc.identifier.issue6
dc.identifier.startpage1819
dc.identifier.endpage1825
dc.contributor.firstauthorID238350


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