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dc.contributor.authorOnur, Rustu
dc.contributor.authorDilekoz, Ergin
dc.contributor.authorGÜLHAN, REZZAN
dc.contributor.authorCarcak, Nihan
dc.contributor.authorSara, Yildirim
dc.contributor.authorOnat, Filiz Yilmaz
dc.date.accessioned2021-03-03T15:44:57Z
dc.date.available2021-03-03T15:44:57Z
dc.identifier.citationCarcak N., Dilekoz E., GÜLHAN R., Onur R., Onat F. Y. , Sara Y., "Increased inhibitory synaptic activity in the hippocampus (CA1) of genetic absence epilepsy rats: Relevance of kindling resistance", EPILEPSY RESEARCH, cilt.126, ss.70-77, 2016
dc.identifier.issn0920-1211
dc.identifier.otherav_4099b495-e6f0-4dd9-8448-8846580f819a
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/47191
dc.identifier.urihttps://doi.org/10.1016/j.eplepsyres.2016.06.013
dc.description.abstractPurpose: Genetic absence epilepsy rats from Strasbourg (GAERS), a well-validated genetic rat model for typical absence epilepsy, are known to manifest a resistance to secondary generalization of abnormal focal electrical activity evoked by kindling. The mechanism of this resistance is still unclear. In order to understand the possible mechanism of kindling resistance, we investigated for the first time, the differences of short-term synaptic plasticity by using a paired-pulse paradigm as an indicator of GABAergic activity in CM region of hippocampus in GAERS and non-epileptic Wistar rats in-vivo.
dc.language.isoeng
dc.subjectTıp
dc.subjectSağlık Bilimleri
dc.subjectDahili Tıp Bilimleri
dc.subjectNöroloji
dc.subjectKLİNİK NEUROLOJİ
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.titleIncreased inhibitory synaptic activity in the hippocampus (CA1) of genetic absence epilepsy rats: Relevance of kindling resistance
dc.typeMakale
dc.relation.journalEPILEPSY RESEARCH
dc.contributor.departmentEskişehir Osmangazi Üniversitesi , ,
dc.identifier.volume126
dc.identifier.startpage70
dc.identifier.endpage77
dc.contributor.firstauthorID82385


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