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Cytotoxic T lymphocyte antigen-4 (CTLA-4) A49G polymorphism and autoimmune blood diseases

dc.contributor.authorHancer, Veysel Sabri
dc.contributor.authorAkturk, Faruk
dc.contributor.authorKucukkaya, Reyhan
dc.date.accessioned2021-03-03T16:26:48Z
dc.date.available2021-03-03T16:26:48Z
dc.date.issued2010
dc.identifier.citationAkturk F., Hancer V. S. , Kucukkaya R., "Cytotoxic T lymphocyte antigen-4 (CTLA-4) A49G polymorphism and autoimmune blood diseases", TURKISH JOURNAL OF HEMATOLOGY, cilt.27, sa.2, ss.78-81, 2010
dc.identifier.issn1300-7777
dc.identifier.othervv_1032021
dc.identifier.otherav_444855b6-f82d-42ef-9f72-726c518df442
dc.identifier.urihttp://hdl.handle.net/20.500.12627/49590
dc.identifier.urihttps://doi.org/10.5152/tjh.2010.04
dc.description.abstractObjective: The cytotoxic T lymphocyte associated antigen-4 (CTLA-4) is expressed on T lymphocytes, and inhibits the T-cell responses. In animal models, it has been shown that complete CTLA-4 deficiency was lethal due to massive infiltration of tissues by polyclonally proliferating lymphocytes. CTLA-4 A49G polymorphism, which has been suggested to reduce the inhibitory function of the CTLA-4 molecule, was found to be associated with various autoimmune diseases in recent studies.
dc.language.isoeng
dc.subjectSağlık Bilimleri
dc.subjectHematoloji
dc.subjectDahili Tıp Bilimleri
dc.subjectTıp
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectHEMATOLOJİ
dc.subjectİç Hastalıkları
dc.titleCytotoxic T lymphocyte antigen-4 (CTLA-4) A49G polymorphism and autoimmune blood diseases
dc.typeMakale
dc.relation.journalTURKISH JOURNAL OF HEMATOLOGY
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume27
dc.identifier.issue2
dc.identifier.startpage78
dc.identifier.endpage81
dc.contributor.firstauthorID196414


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