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dc.contributor.authorAkkaya, Vakur
dc.contributor.authorAydin, Sengul
dc.contributor.authorOzturk, Guzin Zeren
dc.contributor.authorArman, Yucel
dc.contributor.authorKose, Murat
dc.contributor.authorTukek, Tufan
dc.contributor.authorEmet, Samim
dc.contributor.authorOzcan, Mustafa
dc.contributor.authorArslan, Kadem
dc.date.accessioned2021-03-03T17:42:29Z
dc.date.available2021-03-03T17:42:29Z
dc.date.issued2015
dc.identifier.citationOzcan M., Ozturk G. Z. , Kose M., Emet S., Aydin S., Arslan K., Arman Y., Akkaya V., Tukek T., "Evaluation of malnutrition with blood ghrelin and fecal elastase levels in acute decompensated heart failure patients", TURK KARDIYOLOJI DERNEGI ARSIVI-ARCHIVES OF THE TURKISH SOCIETY OF CARDIOLOGY, cilt.43, sa.2, ss.131-137, 2015
dc.identifier.otherav_4b021ee7-c1fa-4dc5-bdc4-211de1de75c8
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/53863
dc.identifier.urihttps://doi.org/10.5543/tkda.2015.06606
dc.description.abstractObjectives: Exocrine pancreatic dysfunction may contribute to malnutrition and lack of appetite in the advanced stages of heart failure. Nutritional assessment was carried out on patients diagnosed with mild or moderate/severe heart failure. Fecal elastase levels are an indicator of pancreatic exocrine function and ghrelin is an appetite hormone which is also investigated for its contribution to malnutrition.
dc.language.isoeng
dc.subjectDahili Tıp Bilimleri
dc.subjectSağlık Bilimleri
dc.subjectKardiyoloji
dc.subjectKlinik Tıp
dc.subjectKlinik Tıp (MED)
dc.subjectTıp
dc.subjectCARDIAC ve CARDIOVASCULAR SİSTEMLER
dc.titleEvaluation of malnutrition with blood ghrelin and fecal elastase levels in acute decompensated heart failure patients
dc.typeMakale
dc.relation.journalTURK KARDIYOLOJI DERNEGI ARSIVI-ARCHIVES OF THE TURKISH SOCIETY OF CARDIOLOGY
dc.contributor.departmentIstanbul Okmeydani Training & Research Hospital , ,
dc.identifier.volume43
dc.identifier.issue2
dc.identifier.startpage131
dc.identifier.endpage137
dc.contributor.firstauthorID221269


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