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Serum levels of tumor necrosis factor-alpha, interleukin-6 and interleukin-8 are not increased in dyspeptic patients with Helicobacter pylori-associated gastritis

dc.contributor.authorDavutoglu, C
dc.contributor.authorBorazan, A
dc.contributor.authorAydemir, S
dc.contributor.authorUstundag, Y
dc.contributor.authorAtmaca, H
dc.contributor.authorBayraktaroglu, T
dc.contributor.authorAras, AS
dc.date.accessioned2021-03-02T20:11:50Z
dc.date.available2021-03-02T20:11:50Z
dc.date.issued2004
dc.identifier.citationBayraktaroglu T., Aras A., Aydemir S., Davutoglu C., Ustundag Y., Atmaca H., Borazan A., "Serum levels of tumor necrosis factor-alpha, interleukin-6 and interleukin-8 are not increased in dyspeptic patients with Helicobacter pylori-associated gastritis", MEDIATORS OF INFLAMMATION, cilt.13, sa.1, ss.25-28, 2004
dc.identifier.issn0962-9351
dc.identifier.otherav_00c75181-e773-42d0-ad18-3b606717a300
dc.identifier.othervv_1032021
dc.identifier.urihttp://hdl.handle.net/20.500.12627/6512
dc.identifier.urihttps://doi.org/10.1080/09629350410001664789
dc.description.abstractIntroduction: Helicobacter pylori ( H. pylori) is a non-invasive microorganism causing intense gastric mucosal inflammatory and immune reaction. H. pylori-induced gastric mucosal cytokine overproduction has been clearly documented previously. The stomach has a large surface area and continuous spill-over of locally produced cytokines into the blood stream is a possibility. There are few and conflicting data on circulatory proinflammatory cytokine levels in patients with H. pylori infection.
dc.language.isoeng
dc.subjectYaşam Bilimleri
dc.subjectMoleküler Biyoloji ve Genetik
dc.subjectTemel Bilimler
dc.subjectSağlık Bilimleri
dc.subjectHistoloji-Embriyoloji
dc.subjectTemel Tıp Bilimleri
dc.subjectTıp
dc.subjectİmmünoloji
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectMoleküler Biyoloji ve Genetik
dc.subjectHÜCRE BİYOLOJİSİ
dc.titleSerum levels of tumor necrosis factor-alpha, interleukin-6 and interleukin-8 are not increased in dyspeptic patients with Helicobacter pylori-associated gastritis
dc.typeMakale
dc.relation.journalMEDIATORS OF INFLAMMATION
dc.contributor.department, ,
dc.identifier.volume13
dc.identifier.issue1
dc.identifier.startpage25
dc.identifier.endpage28
dc.contributor.firstauthorID170898


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