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dc.contributor.authorTAMER-TOPTANI, S
dc.contributor.authorAYKAC-TOKER, G
dc.contributor.authorUysal, Mehmet Müjdat
dc.contributor.authorKocak-Toker, N
dc.contributor.authorAKDENIZ, S
dc.contributor.authorUGURNAL, B
dc.contributor.authorDogru-Abbasoglu, Semra
dc.date.accessioned2021-03-04T10:25:10Z
dc.date.available2021-03-04T10:25:10Z
dc.date.issued1998
dc.identifier.citationDogru-Abbasoglu S., UGURNAL B., TAMER-TOPTANI S., AKDENIZ S., AYKAC-TOKER G., Kocak-Toker N., Uysal M. M. , "Mitochondrial lipid peroxidation and antioxidant system in aged rats", ARCHIVES OF GERONTOLOGY AND GERIATRICS, cilt.27, sa.1, ss.35-40, 1998
dc.identifier.issn0167-4943
dc.identifier.othervv_1032021
dc.identifier.otherav_6d1d41d1-4692-418b-a08f-9940fea23eb5
dc.identifier.urihttp://hdl.handle.net/20.500.12627/75390
dc.identifier.urihttps://doi.org/10.1016/s0167-4943(98)00015-6
dc.description.abstractMitochondria are especially important in cellular senescence since reactive oxygen species (ROS) have been found to be generated constantly as an endogen threat. On the other hand, mitochondrial defence depends mainly on glutathione (GSH), superoxide dismutase (SOD) and glutathione peroxidase (GP(x)). In this study, liver and brain mitochondria of 6- and 22-month-old rats were investigated. Liver mitochondrial malondialdehyde (MDA) levels were found increased, whereas mitochondrial GSH was decreased. SOD activity has been observed elevated, whereas GP(x) was unchanged. However, brain mitochondrial MDA, GSH and SOD and GP(x) activites were found unchanged in old rats. (C) 1998 Elsevier Science Ireland Ltd. All rights reserved.
dc.language.isoeng
dc.subjectİç Hastalıkları
dc.subjectDahili Tıp Bilimleri
dc.subjectGeriatri
dc.subjectKlinik Tıp (MED)
dc.subjectTıp
dc.subjectSağlık Bilimleri
dc.subjectGERİATRİK VE GERONTOLOJİ
dc.subjectKlinik Tıp
dc.titleMitochondrial lipid peroxidation and antioxidant system in aged rats
dc.typeMakale
dc.relation.journalARCHIVES OF GERONTOLOGY AND GERIATRICS
dc.contributor.department, ,
dc.identifier.volume27
dc.identifier.issue1
dc.identifier.startpage35
dc.identifier.endpage40
dc.contributor.firstauthorID16969


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