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dc.contributor.authorOz, Buge
dc.contributor.authorIsler, Cihan
dc.contributor.authorUzan, Mustafa
dc.contributor.authorTanriverdi, Taner
dc.contributor.authorUlu, Mustafa Onur
dc.contributor.authorERASLAN, Berna Senel
dc.contributor.authorOzyurt, Emin
dc.contributor.authorOzkara, Cigdem
dc.contributor.authorBiceroglu, Hueseyin
dc.date.accessioned2021-03-02T20:36:57Z
dc.date.available2021-03-02T20:36:57Z
dc.date.issued2010
dc.identifier.citationUlu M. O. , Tanriverdi T., Oz B., Biceroglu H., Isler C., ERASLAN B. S. , Ozkara C., Ozyurt E., Uzan M., "The expression of astroglial glutamate transporters in patients with focal cortical dysplasia: an immunohistochemical study", ACTA NEUROCHIRURGICA, cilt.152, sa.5, ss.845-853, 2010
dc.identifier.issn0001-6268
dc.identifier.othervv_1032021
dc.identifier.otherav_02d633c8-6a61-4b11-8560-84f847a63d8d
dc.identifier.urihttp://hdl.handle.net/20.500.12627/7857
dc.identifier.urihttps://doi.org/10.1007/s00701-009-0548-2
dc.description.abstractAn abnormal increase in the extracellular glutamate is thought to play a crucial role in the initiation, spread, and maintenance of seizure activity.In normal conditions, the majority of this excess glutamate is cleared via glial glutamate transporters (EAAT-1 and EAAT-2). We aimed to examine the immunohistochemical expression of these transporters in the dysplastic tissues of patients with focal cortical dysplasia (FCD).
dc.language.isoeng
dc.subjectCerrahi Tıp Bilimleri
dc.subjectTıp
dc.subjectNöroloji
dc.subjectSağlık Bilimleri
dc.subjectDahili Tıp Bilimleri
dc.subjectCERRAHİ
dc.subjectKlinik Tıp (MED)
dc.subjectKlinik Tıp
dc.subjectKLİNİK NEUROLOJİ
dc.titleThe expression of astroglial glutamate transporters in patients with focal cortical dysplasia: an immunohistochemical study
dc.typeMakale
dc.relation.journalACTA NEUROCHIRURGICA
dc.contributor.department, ,
dc.identifier.volume152
dc.identifier.issue5
dc.identifier.startpage845
dc.identifier.endpage853
dc.contributor.firstauthorID33052


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