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dc.contributor.authorYilmazer, Selma
dc.contributor.authorGezen-Ak, Duygu
dc.contributor.authorDursun, Erdinc
dc.date.accessioned2021-03-04T11:58:43Z
dc.date.available2021-03-04T11:58:43Z
dc.date.issued2011
dc.identifier.citationDursun E., Gezen-Ak D., Yilmazer S., "A Novel Perspective for Alzheimer's Disease: Vitamin D Receptor Suppression by Amyloid-beta and Preventing the Amyloid-beta Induced Alterations by Vitamin D in Cortical Neurons", JOURNAL OF ALZHEIMERS DISEASE, cilt.23, sa.2, ss.207-219, 2011
dc.identifier.issn1387-2877
dc.identifier.othervv_1032021
dc.identifier.otherav_750c58be-9b63-4ac8-a17d-08b7862c7200
dc.identifier.urihttp://hdl.handle.net/20.500.12627/80422
dc.identifier.urihttps://doi.org/10.3233/jad-2010-101377
dc.description.abstractAmyloid-beta (A beta) is the core component of amyloid plaques of Alzheimer's disease (AD). The effects of A beta include damage to neuronal plasma membrane, disruption of Ca2+ homeostasis, and alterations of neurotrophic factor levels. The aim of this study was to determine the effects of A beta treatment on vitamin D receptor (VDR), L-type voltage sensitive calcium channels A1C (LVSCC A1C), NGF, and observing the effects of vitamin D treatment on A beta induced alterations in primary cortical neurons. As to the latter, we aimed to test the suggested neuroprotective role of vitamin D as a neglected neurosteroid.
dc.language.isoeng
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectTemel Bilimler
dc.subjectNEUROSCIENCES
dc.subjectSinirbilim ve Davranış
dc.subjectYaşam Bilimleri
dc.titleA Novel Perspective for Alzheimer's Disease: Vitamin D Receptor Suppression by Amyloid-beta and Preventing the Amyloid-beta Induced Alterations by Vitamin D in Cortical Neurons
dc.typeMakale
dc.relation.journalJOURNAL OF ALZHEIMERS DISEASE
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume23
dc.identifier.issue2
dc.identifier.startpage207
dc.identifier.endpage219
dc.contributor.firstauthorID6276


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