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dc.contributor.authorSeckin, Ismail
dc.contributor.authorTunali, Gulden
dc.contributor.authorTanriverdi, Gamze
dc.contributor.authorAslan, Esra Guler
dc.contributor.authorOzdemir, Semra
dc.contributor.authorOzturk, Bahar
dc.contributor.authorAYLA, ŞULE
dc.contributor.authorBilgic, Bulent E.
dc.contributor.authorSofuoglu, Kenan
dc.contributor.authorOzdemir, A. Arman
dc.contributor.authorSONER, BURAK CEM
dc.contributor.authorKarahuseyinoglu, Sercin
dc.date.accessioned2021-03-04T18:04:54Z
dc.date.available2021-03-04T18:04:54Z
dc.date.issued2018
dc.identifier.citationAYLA Ş., Tunali G., Bilgic B. E. , Sofuoglu K., Ozdemir A. A. , Tanriverdi G., Ozdemir S., SONER B. C. , Ozturk B., Karahuseyinoglu S., et al., "Antioxidant activity of CAPE (caffeic acid phenethyl ester) in vitro can protect human sperm deoxyribonucleic acid from oxidative damage", ACTA HISTOCHEMICA, cilt.120, ss.117-121, 2018
dc.identifier.issn0065-1281
dc.identifier.othervv_1032021
dc.identifier.otherav_88d39066-0d80-4ecb-a261-e7ed01abfa1c
dc.identifier.urihttp://hdl.handle.net/20.500.12627/92824
dc.identifier.urihttps://doi.org/10.1016/j.acthis.2018.01.001
dc.description.abstractPurpose: Sperm processing (e.g., centrifugation) used in preparation for assisted reproduction can result in excessive generation of reactive oxygen species (ROS) and potential sperm damage. The use of antioxidants during sperm processing has been shown to prevent iatrogenic sperm damage, including DNA damage. In this study, we evaluated the effect of caffeic acid phenethyl ester (CAPE) on oxidative stress mediated sperm dysfunction and DNA damage.
dc.language.isoeng
dc.subjectMoleküler Biyoloji ve Genetik
dc.subjectTemel Bilimler
dc.subjectTemel Tıp Bilimleri
dc.subjectHistoloji-Embriyoloji
dc.subjectSağlık Bilimleri
dc.subjectTıp
dc.subjectYaşam Bilimleri (LIFE)
dc.subjectMoleküler Biyoloji ve Genetik
dc.subjectHÜCRE BİYOLOJİSİ
dc.subjectYaşam Bilimleri
dc.titleAntioxidant activity of CAPE (caffeic acid phenethyl ester) in vitro can protect human sperm deoxyribonucleic acid from oxidative damage
dc.typeMakale
dc.relation.journalACTA HISTOCHEMICA
dc.contributor.departmentİstanbul Üniversitesi , ,
dc.identifier.volume120
dc.identifier.issue2
dc.identifier.startpage117
dc.identifier.endpage121
dc.contributor.firstauthorID250613


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